Since the mid 1960’s, Deca Durabolion has become a staple in most testosterone and/or dianabol cycles, the main reason is that Deca adds a lot of strength to a cycle without increasing side effects.
When used in a testosterone and/or dianabol cycle, deca really adds weight to the cycle without much more stress on the system. Deca does not add additional side-effects when used with other steroids, but it should never be used alone. Standalone use of nandrolone comes with its very own set of nasty side-effects, the most infamous of them all is the dreaded “Deca Dick”, where the affected user is unable to get or sustain an erection.
This is due to the fact that without the stronger testosterone metabolites like dihydrotestosterone (DHT), the softer and much weaker metabolites from deca will flood your receptors, not leaving room for stronger androgens. Not only are deca’s weaker metabolites an issue when there is an absence of stronger androngens, but nandrolone itself can, and will, attach to progesterone receptors. This causes side effects that only affect the user in the absence of supraphysiological amounts of stronger androgens.
The best way to explain deca durabolin is that it's basically a progesterone-like hormone that builds muscle mass, strength and, to a lesser degree, helps repair joints. It should not give you any bad side effects when used with stronger androgens that will mask and counteract possible problems from nandrolone. If ever used by itself, the bad side-effects from nandrolone will crush your libido, sexual desire, cause bloating and gynecomastia (bitch tits).
Nandrolone decanoate shares the actions of endogenous androgens such as testosterone. Exogenous androgens such as nandrolone decanoate promote protein anabolism and stimulate appetite which results in a reversal of catabolic processes and negative nitrogen balance. Increases in lean body mass in patients with cachexia (e.g., malnourished dialysis patients) and decreased bone resorption and increased bone density in patients with osteoporosis are often noted. Blood glucose, erythrocyte production, and the balance of calcium are also affected by androgens. Increased erythrocyte production is apparently due to enhanced production of erythropoietic stimulating factor. Patients with anemia associated with renal disease will have increases in red blood cell volume and hemoglobin after receiving nandrolone decanoate.
Since nandrolone decanoate has actions similar to endogenous androgens, administration of nandrolone decanoate has the possibility of causing serious disturbances of growth and sexual development if given to young children and causing unwanted adverse effects in women. Exogenous androgens suppress gonadotropin-releasing hormone, thereby reducing the gonadotropic function of the pituitary through a negative-feedback mechanism. This results in a reduction of endogenous testosterone, luteinizing hormone, and follicle-stimulating hormone. Exogenous androgens may also have a direct effect on the testes. Reversible increases in low-density lipoproteins (LDL) and decreases in high-density lipoproteins (HDL) also occur.
Since deca durabolin is formulated using nandrolone decanoate, one of the longest esters in existence, it is very oil soluble, the way all steroids with long esters are. This easy solubility in oils means that manufacturers have to use a lower percentage of solvents to manufacture the products; thus, making for a less irritating shot. Generally, deca injections are done weekly by most bodybuilders.
By adding 400mg per week of nandrolone to a testosterone cycle or a dianabol cycle, you'll be able to increase your overall steroid dosage, without increasing the side effects. For example, a bodybuilder taking 500mg Sustanon 250 per week, who felt he needed more power in his cycle, would find himself with more side-effects if he were to just take more of the same testosterone. Since testosterone aromatizes at an increasing rate at higher doses, there would be an exponential increase in the likeliness of gynecomastia and water retention if more testosterone was added weekly. When adding nandrolone decanoate to the same testosterone cycle, you are increasing the total amounts of steroids your body is receiving every week. However, you are not adding anymore viable substrate for undesired enzymatic reactions from aromatase and 5-reductase. In simple terms, you use more steroids without more side effects.
The dosage for men is around 400-600mgs per week but that varies depending on goals. I've seen guys use as little as 200mgs per week as a booster, and up to 1500mgs per week as a heavy bulker.
For women, I wouldn't suggest using deca durabolin because of the side effects that are too variable from female to female.
The adverse effects of Nandrolone Decanoate (Deca Durabolin) related to cholesterol levels in the human body results from all other anabolic steroids.
This include increases of LDL the bad cholesterol as well as the reduction of HDL the good cholesterol.
These deviations result in an increased arteriosclerosis risk. Dosage excesses determine the degree to which these fluctuations bring about destructive modifications.
Usage duration and the route of administration are other aspects that affect these negative cholesterol fluctuations.
Deca Durabolin validates in medical studies that when test subjects receive an administration of six hundred milligrams per week for a ten-week period that there is a twenty-six percent drop in HDL or good cholesterol levels.
This may be reduced by stacking with grape seed extract.Nandrolone administration prompts significantly greater negative variations in cholesterol values than that of Testosterone in comparison to the monitoring of HDL or the good cholesterol and LDL or the bad cholesterol levels in a different study with the administration of Testosterone Cypionate.
Since Nandrlone is not an oral C17 Alpha Alkylated anabolic steroid, there are no hepatotoxicity risks.
Hepatotoxicity is chemical induced liver damage, but you do not have to worry about this side effect with Nandrolone Decanoate use.
Change in sex drive or performance; diarrhea; hair loss; headache; trouble sleeping. This list may not describe all possible side effects. Call your health care provider immediately if you are experiencing any signs of an allergic reaction: allergic reactions like skin rash, itching or hives, swelling of the face, lips, or tongue; breast lump; breathing problems; changes in mood, especially anger, depression, or rage; change in voice; dark urine; increase in facial hair; irregular menstrual periods; acne, nausea, vomiting; stomach pain; swelling in ankles or legs; trouble passing urine or change in the amount of urine; yellowing of the eyes or skin.
Menstrual irregularity can occur with nandrolone decanoate therapy in females. Disruption of the regular menstrual cycle secondary to nandrolone decanoate-induced suppression of gonadotropin secretion can lead to amenorrhea or oligomenorrhea.
When androgens (such as nandrolone) are given to women, virilization, manifested by acne, hirsutism, clitoromegaly, male pattern baldness, reduced breast size, and deepening of the voice or hoarseness, can occur. If treatment is discontinued when these symptoms first appear, they usually subside. Prolonged treatment can lead to irreversible masculinity, so the benefit of treatment should be measured against the risk.
Androgens can cause teratogenesis. Androgens are classified as pregnancy category X, and are absolutely contraindicated during pregnancy because of probable adverse effects on the fetus. Androgenic anabolic steroids such as nandrolone decanoate are known to cause embryotoxicity, fetotoxicity, and masculinization of female animal offspring. Nandrolone decanoate is contraindicated in women who are or may become pregnant.
Male patients can experience feminization during prolonged therapy with nandrolone decanoate, which is believed to result from inhibition of gonadotropin secretion and conversion of androgens to estrogens. These effects are more pronounced in patients with concurrent hepatic disease and include mastalgia and gynecomastia. Feminizing effects are generally reversible. Inhibition of testicular function, testicular atrophy, impotence (erectile dysfunction), epididymitis, and bladder irritation can also occur.
Priapism and excessive sexual stimulation, more common in geriatric males, are generally the effect of excessive nandrolone decanoate dosage. Oligospermia and decreased ejaculate volume may occur in patients receiving long-term therapy or excessive doses. Alopecia resembling male pattern baldness has also occurred.
Prostate cancer as a secondary malignancy or prostatic hypertrophy can develop during prolonged therapy with nandrolone decanoate and are more likely to occur in elderly males. Signs of acute epididymitis (e.g., fever, chills, pain in the inguinal region) and/or urinary urgency should prompt withdrawal of the drug and reevaluation of dosage.
When androgens (such as nandrolone) are used in the treatment of immature males, early virilism can be a disadvantage because it is accompanied by premature epiphyseal closure. Monitoring of skeletal maturation should be undertaken at about 6-month intervals. Once the epiphyses have closed, growth is terminated. Even after discontinuation of treatment, epiphyseal closure can be enhanced for several months. Penile enlargement and an increased frequency of erections can also occur.
Peripheral edema can occur with nandrolone use as the result of increased fluid retention (in association with sodium retention) and is manifested by weight gain. In the treatment of patients with impaired renal function or congestive heart failure, the fluid retention is of greater significance. Other serum electrolytes (i.e., calcium, chloride, phosphate, and potassium) are also retained.
Androgen therapy (such as nandrolone) is related to growth and secretion of the sebaceous glands, which can cause an acneiform rash indistinguishable from acne vulgaris.
Hepatic dysfunction can occur from use of androgenic anabolic steroids (such as nandrolone) and have been shown to be more significant with administration of the oral 17-alpha-alkylandrogens (e.g., methyltestosterone). Cholestatic jaundice with, rarely, hepatic necrosis and death have been reported. Elevated hepatic enzymes are more common than overt jaundice.
The drug should be discontinued if cholestatic jaundice or hepatitis occurs. Peliosis hepatis, a condition characterized by splenic tissue being replaced by blood filled cysts, has occurred in patients receiving androgenic anabolic steroids. The cysts are sometimes present with minimal hepatic dysfunction, but may be associated with hepatic failure. They are often not noticeable until life-threatening hepatic failure or intra-abdominal hemorrhage develops.
Discontinuation of steroid therapy usually results in complete disappearance of cysts. Hepatoma also occurs rarely and is usually benign and androgen-dependent; life-threatening malignant hepatoma has been reported. Withdrawal of drug often results in regression or cessation of progression of the tumors. However, hepatomas associated with androgens or anabolic steroids are much more vascular than other hepatic tumors and may be undetected until life-threatening intra-abdominal hemorrhage develops.
Androgen therapy (such as nandrolone) has induced osteolysis and can exacerbate hypercalcemia. Androgen-induced hypercalcemia occurs especially in immobile patients and those with metastatic carcinoma of the breast.
Observational studies in post-menopausal women, bodybuilders, and weightlifters using anabolic steroids have revealed 'pro-atherogenic' changes in lipid profiles, including decreases in HDL concentrations and increases in LDL concentrations. Synthetic androgens may produce a greater lowering of the HDL-C:LDL-C ratio than does testosterone. Oral anabolic steroids (e.g., stanozolol) may produce greater changes than parenteral ones. Although the implications of androgen-induced (such as nandrolone) hypercholesterolemia are unclear, caution should be exercised, particularly in patients predisposed to dyslipidemias or atherosclerosis.
Androgen therapy (such as nandrolone) can produce libido decrease or libido increase. Geriatric males have been found to be more likely to experience excessive sexual stimulation.
Miscellaneous adverse reactions to nandrolone decanoate therapy have included decreased glucose tolerance, diarrhea, edema, excitability, habituation, increased CPK and creatinine, insomnia, mental depression, nausea and vomiting.
Intramuscular administration of anabolic steroids (such as nandrolone) can cause inflammation, urticaria, postinjection induration and furunculosis. Patients should be observed for any signs of an injection site reaction.
Anabolic steroids (such as nandrolone) may cause suppression of clotting factors II, V, VII, and X. Prolonged bleeding time may occur.
This list may not include all possible adverse reactions or side effects. Call your health care provider immediately if you are experiencing any signs of an allergic reaction: skin rash, itching or hives, swelling of the face, lips, or tongue, blue tint to skin, chest tightness, pain, difficulty breathing, wheezing, dizziness, red, a swollen painful area/areas on the leg.
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